There's a fine line between skin care that makes you look dewy and skin care that makes you look (and feel) greasy. Sunscreen often straddles that fine line. Some formulas leave a powdery white cast on deeper complexions; others make your pores look like they've been dunked in a deep frier. Neither of those side effects is cute, but what's really not cute is skipping out on protecting your skin from the sun — so finding the right sunscreen is crucial.

Charles Leclerc saw off team-mate Carlos Sainz by just 0.067 seconds as Ferrari locked out the front row for Sunday’s Mexican Grand Prix. Max Verstappen improved on his final run at the Autodromo Hermanos Rodriguez but could not usurp the Ferrari drivers, finishing 0.097 sec adrift. The triple world champion also faces a nervous wait to see if he is moved down the grid after being summoned to see the stewards. Verstappen, charged with impeding on the pit exit, is among four drivers who will be hauled in front of the race officials. Lewis Hamilton, disqualified from finishing runner-up to Verstappen at the United States Grand Prix a week ago, may too feel the wrath of the race referees for failing to slow under yellow flags. Hamilton finished only sixth, 0.288 sec back. George Russell, who qualified eighth, and Fernando Alonso, 13th on the grid, have also been called to see the stewards for blocking on the pit exit in Q1. Verstappen hit the kerb at Turn 8 in his first attempt in Q3 to leaving him trailing Leclerc by 0.120 sec. The Red Bull man, who has won 15 of the 18 rounds so far, produced a better last lap, but could not prevent Leclerc from sealing his second pole in as many weekends. “I didn’t expect to be one pole because we looked to be lacking quite a bit of pace after practice,” said Leclerc. “But for some reason once we put everything together it went well and on the new tyres we gained a lot. “I’m already focusing on tomorrow’s race because we have had many pole positions this season, but we need to convert it into victory and that is going to be very difficult.” Nearly 400,000 spectators will pass through the gates at the high-altitude Mexico City venue this weekend with the majority here to support Sergio Perez. But the home favourite failed to deliver, finishing nearly three tenths adrift of Verstappen and qualified fifth, one position behind Daniel Ricciardo who impressed in his AlphaTauri. Earlier, Lando Norris was the surprise name eliminated from the opening phase of qualifying, leaving the in-form British driver in 19th place. Norris, who has finished on the podium at the past four races, attempted to progress from Q1 on the slower medium rubber in order to save a set of speedier softs. But the plan backfired when Norris’ lap wasn’t quick enough. Norris bolted on the soft tyres but then made a mistake a Turn 10. He aborted the lap and prepared for one last attempt, only to run into yellow flags at the opening bend after Alonso spun in his Aston Martin. Norris’ qualifying was over leaving him a tall order to salvage anything from tomorrow’s race. American rookie Logan Sargeant, who earned his first point in F1 last weekend in Austin, will prop up the grid after he saw two laps scrubbed off by the stewards for exceeding track limits. Read More Essex boy with Italian twang – History-maker Ollie Bearman impresses in Mexico Max Verstappen sets fastest time in Mexican Grand Prix practice Max Verstappen urges fans to show him respect ahead of feisty Mexican Grand Prix Lewis Hamilton claims many more cars were illegal at United States Grand Prix Max Verstappen beefs up security in preparation for hostile reception in Mexico On this day in 2015: Lewis Hamilton crowned F1 world champion for third time

India and England have had very different experiences at the 2023 Cricket World Cup. For

The brain’s appetite control centre is different in the brains of people who are overweight or live with obesity, a new study suggests. The researchers said their findings add further evidence to the relevance of brain structure to weight and food consumption. Current estimates suggest that more than 1.9 billion people worldwide are either overweight or obese. And, according to the Office for Health Improvement and Disparities, almost two-thirds of adults in the UK are overweight or living with obesity. This increases the risk of developing health issues like type 2 diabetes, heart disease and stroke, cancer and poorer mental health. The last two decades have given us important insights about appetite control and how it may be altered in obesity Professor Paul Fletcher, University of Cambridge A number of factors influence how much people eat and what they eat, including genetics, hormone regulation, and the environment they live in, researchers say. However, it is not entirely clear what happens to the brain to tell us that we are hungry or full. Past studies have shown that the hypothalamus – a small region of the brain about the size of an almond – plays an important role. Dr Stephanie Brown, from the Department of Psychiatry and Lucy Cavendish College, University of Cambridge, said: “Although we know the hypothalamus is important for determining how much we eat, we actually have very little direct information about this brain region in living humans. “That’s because it is very small and hard to make out on traditional MRI brain scans.” Professor Paul Fletcher, the study’s senior author, from the Department of Psychiatry and Clare College, Cambridge, said: “The last two decades have given us important insights about appetite control and how it may be altered in obesity. “Metabolic researchers at Cambridge have played a leading role in this. “Our hope is that, by taking this new approach to analysing brain scans in large datasets, we can further extend this work into humans, ultimately relating these subtle structural brain findings to changes in appetite and eating and generating a more comprehensive understanding of obesity.” The majority of evidence for the role of the hypothalamus in appetite regulation comes from animal studies, which indicate complex interacting pathways within the hypothalamus, with different cell populations acting together to tell us when we are hungry or full. To get around this researchers used an algorithm developed using machine learning to analyse brain scans taken from 1,351 young adults across a range of BMI scores. They looked for differences in the hypothalamus when comparing individuals who are underweight, healthy weight, overweight, or living with obesity. According to the findings, the overall volume of the hypothalamus was significantly larger in the overweight and obese groups of young adults. The researchers describe a significant relationship between volume of the hypothalamus and body mass index (BMI). The differences were most apparent in those sub-regions of the hypothalamus that control appetite through the release of hormones to balance hunger and fullness. While the exact significance of the finding is unclear, one explanation is that the change relates to inflammation, the researchers suggest. Eating a high-fat diet could trigger inflammation of our appetite control centre Dr Stephanie Brown, University of Cambridge Previous animal studies have shown that a high-fat diet can cause inflammation of the hypothalamus, which in turn prompts insulin resistance and obesity. In mice, just three days of a fat-rich diet is enough to cause this inflammation. Other studies have shown that this inflammation can raise the threshold at which animals are full – in other words, they have to eat more food than usual to feel full. Dr Brown added: “If what we see in mice is the case in people, then eating a high-fat diet could trigger inflammation of our appetite control centre. “Over time, this would change our ability to tell when we’ve eaten enough and to how our body processes blood sugar, leading us to put on weight.” More research is needed to confirm whether increased volume in the hypothalamus is a result of being overweight or whether people with larger hypothalami are predisposed to eat more in the first place. It is also possible that these two factors interact with each other, causing a feedback loop, the study published in Neuroimage: Clinical, and supported by the Bernard Wolfe Health Neuroscience Fund, Wellcome and the NIHR Cambridge Biomedical Research Centre, with additional funding from Alzheimer’s Research UK, suggests.

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